In the White lab at UCLA I study the role of activity-dependent transcriptional regulation in learned vocal communication using the zebra finch model system. Given recent advances in zebra finch genetics, and  that over 20 human intellectual disability genes are dynamically regulated by learned vocalization in songbirds, the zebra finch system is uniquely poised to contribute new gains in drug discovery for helping human patients with communication deficits. My work specifically focuses on understanding the mechanistic basis for a novel drug that targets epigenetic mechanisms in cognition.